BioOncology Watch

Timely Information for Practicing Physicians

 

december 2003

ERYTHROPOIETIN

Treatment of anemia in patients with head and neck cancer.  Michael Henke et al reported a randomized, double-blind, placebo-controlled trial to determine if erythropoietin could improve cancer control and survival in 351 patients irradiated for head and neck cancer.  Eligible radiotherapy patients (blood hemoglobin concentrations <120 g/L for women and <130 g/L for men) were randomized to receive either epoetin b 300 IU/kg (n = 180) or placebo (n = 171) 3 times weekly beginning 10-14 days before and continuing throughout radiotherapy.  Patients were stratified by tumor resection status: stratum 1, completely resected; stratum 2, incompletely resected; stratum 3, unresectable.  Locoregional progression-free survival was poorer in epoetin b-treated patients than in placebo-treated patients. Analysis by stratum revealed that locoregional progression-free survival was similar between the treatment groups for stratum 1 patients, but stratum-2 and -3 patients fared worse when given epoetin b than when given placebo.  Overall survival decreased in the epoetin b treatment group compared to the placebo group. These findings demonstrated that while anemia was corrected by epoetin b therapy, disease control was impaired in epoetin b-treated patients with head and neck cancer undergoing irradiation.  These findings are consistent with recent observations of the BEST Investigators and Study Group (Leyland-Jones. Lancet Oncology 2003;4:459-460 [letter]).  An Independent Data Monitoring Committee terminated this trial early because metastatic breast cancer patients given erythropoietin plus first-line chemotherapy were found to have lower 12-month survival rates than patients receiving placebo plus first-line chemotherapy.  (Henke M, et al. Lancet 2003;262:1255-1260)

 

RITUXIMAB

Mechanism of action.  Wen-Kai Weng and Ronald Levy performed in vitro antibody-dependent cellular cytotoxicity (ADCC) assays on pretreatment tumor cells from 87 patients with follicular lymphoma subsequently treated with rituximab between 1993 and 2003.  No difference in susceptibility of tumor cells to rituximab-mediated ADCC was found between patients who did or did not respond to rituximab therapy.  However, subgroup analyses showed that both the FcgRIIIa 158 valine/valine and FcgRIIa 131 histidine/histidine genotypes were independently associated with clinical responses to rituximab and increased progression-free survival. This study supports the notion that ADCC plays an important role in the clinical effect of rituximab.  Analysis of Fc receptor polymorphisms in future trials of rituximab therapy is needed.  (Weng W-K and Levy R. J Clin Oncol 2003; 21:3940-3947)

 

Downmodulation of CD20 in chronic lymphocytic leukemia (CLL) patients.  Iman Jiliani and colleagues analyzed peripheral blood samples from 65 patients with CLL treated with rituximab and demonstrated that rituximab was detectable on the lymphocytes of only 3 patients despite the fact that most patients had become CD20-negative.  However, rituximab binding to the CD20 antigen on the surface of CLL cells was detectable when CLL cells were mixed with rituximab.  Rituximab subsequently became undetectable when plasma was added due to a down-modulation of the expression of both CD20 antigen and mRNA. The CLL cells regained CD20 expression after 24 hours in culture.  These experiments indicated that rituximab did not mask expression of CD20 on CLL cells.  Instead, rituximab therapy, in the presence of plasma, was observed to cause a transient down-modulation of CD20 expression on CLL cells. Further studies are needed to elucidate the importance of this finding.  Extrapolation of these results to other lymphomas cannot be done at this time due to a lack of data.  (Jiliani I, et al. Blood 2003;102:3514-3520)

 

TRASTUZUMAB

Every 3 weeks administration in combination with paclitaxel.  Brian Leyland-Jones et al evaluated the pharmacokinetics and safety of trastuzumab plus paclitaxel given every 3 weeks to 32 women with HER-2-overexpressing metastatic breast cancer.  Patients received paclitaxel 175 mg/m2 i.v. on Day 0 and a loading dose of 8 mg/kg of trastuzumab on Day 1. Thereafter, trastuzumab 6 mg/kg and paclitaxel 175 mg/m2 were administered i.v. every 3 weeks for up to 7 cycles. Trastuzumab-related adverse events included infusion reactions and cardiac dysfunction, and ≥ 15% decreases in ejection fraction occurred in 10 patients. Objective responses were achieved in 59% of patients and 22% had stable disease.  Median duration of response was 10.5 months; median time to progression was 12.2 months. Thus, administration of trastuzumab every 3 weeks in combination with paclitaxel resulted in clinical response rates that compared favorably with those of standard weekly trastuzumab plus chemotherapy.  Further study of higher-dose 3-week trastuzumab regimens is warranted. (Leyland-Jones B, et al. J Clin Oncol 2003;21:3965-3971)

 

IMATINIB MESYLATE

Risk and prognosis of central nervous system (CNS) leukemia.  Heike Pfeifer et al analyzed 107 patients with relapsed or refractory Philadelphia chromosome (Ph)-positive acute lymphoid leukemia (ALL; n = 65) or chronic myeloid leukemia (CML) in blast crisis (n = 42) who received imatinib therapy and did not receive prophylactic intrathecal chemotherapy.  Among these patients, 12% developed CNS leukemia.  Twelve patients had a lymphoid or biphenotypic phenotype and 1 patient had CML in myeloid blast crisis.  In addition, patients who received prior prophylactic cranial irradiation did not develop meningeal leukemia.  Two of 3 patients who presented with meningeal leukemia achieved prolonged molecular remissions with imatinib, cranial irradiation, and intrathecal chemotherapy.  There were no long-term survivors among patients who had combined CNS and systemic leukemia.  These data indicate that imatinib-treated patients with Ph-positive ALL or CML in lymphoid blast crisis are at risk for CNS leukemia and should receive CNS prophylaxis.  (Pfeifer H, et al. Clin Cancer Res 2003;9:4674-4681)

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