BioOncology Watch

Timely Information for Practicing Physicians

 

may 2003

NON-HODGKIN'S LYMPHOMA (NHL)

Safety profile of Zevalin®.  Zevalin (90Y ibritumomab tiuxetan; IDEC Pharmaceuticals) is a 90Y-labeled anti-CD20 antibody.  Thomas Witzig and colleagues presented safety data obtained from 5 studies (N = 349) of Zevalin treatment of patients with relapsed or refractory low-grade, follicular, or transformed CD20-positive B-cell NHL.  Patients were administered rituximab on Days 1 and 8 and either 0.4 mCi/kg (patients with platelet counts ≥150,000/mL) or 0.3 mCi/kg (patients with platelet counts between 100,000 and 149,000/mL) of Zevalin on Day 8 (maximum dose, 32 mCi).  Grade 3 and grade 4 (National Cancer Institute's Common Toxicity Criteria) neutropenia, thrombocytopenia, and anemia developed in 30% and 30%, 53% and 10%, and 13% and 4% of patients, respectively.  Patients with bone marrow involvement with lymphoma (n = 146 [42%]) had a greater incidence of grade 4 neutropenia (p = 0.001), thrombocytopenia (p = 0.013), and anemia (p = 0.040).  Nadir counts occurred at week 7-9 and lasted 1-4 weeks.  Seven percent of patients were hospitalized with infection and 2% of patients had grade 3 or 4 bleeding adverse events.  Rituximab infusion-related toxicities were typically grade 1 or 2.  No significant organ toxicity was observed.  Myelodysplasia or acute myelogenous leukemia was reported in 5 patients (1%) from 8 to 34 months after treatment.  These data demonstrate that Zevalin has an acceptable safety profile in patients with relapsed or refractory NHL with less than 25% lymphoma marrow involvement, adequate marrow reserve, platelet counts greater than 100,000/mL, and neutrophils greater than 1,500 cells/mL.  (Witzig TE, et al. J Clin Oncol 2003;21:1263-1270)

 

PANCREATIC CANCER

Phase II study of the farnesyltransferase inhibitor R115777.  Steven Cohen and associates conducted a study in which 20 patients with previously untreated measurable metastatic pancreatic cancer were administered 300 mg of R115777, a selective inhibitor of farnesyltransferase, orally every 12 hours for 21 of 28 days.  R115777 had previously demonstrated activity against pancreatic cancer cell lines and xenograft models.  Farnesyltransferase activity in peripheral-blood mononuclear cells of patients was found to decrease by a mean of 49.8% ± 9.8% 4 hours after the initial dose of R115777.  Despite this inhibition of farnesyltransferase activity, no objective responses were achieved.  The median time to progression was 4.9 weeks and the median overall survival time was 19.7 weeks.  Grade 3 or 4 toxicities included transaminase elevation, anemia, neutropenia, thrombocytopenia, fatigue, nausea/vomiting, rash, and anorexia and 9 (45%) patients required dose reductions of R115777 during the first cycle.  This study did not demonstrate single-agent R115777 clinical activity against previously untreated metastatic pancreatic cancer. (Cohen SJ, et al. J Clin Oncol 2003;21:1301-1306)

 

RENAL CELL CARCINOMA (RCC)

Adjuvant interferon alfa therapy.  Edward Messing and others performed a phase III study in which 283 patients with locally and/or regionally extensive RCC who had undergone nephrectomy and lymphadenectomy were randomized to adjuvant interferon alfa-NL (Wellferon; Burroughs-Wellcome) therapy or observation.  Interferon alfa-NL was administered by intramuscular injection daily for 5 days every 3 weeks for up to 12 cycles by the following schedule: 3 million U/m2 on Day 1; 5 million U/m2 on Day 2, and 20 million U/m2 on Days 3, 4, and 5. With a median follow-up of 10.4 years, the median recurrence-free survival was 3.0 years for the observation group compared to 2.2 years for interferon-treated patients (p = 0.33).  The median overall survival times were 7.4 years and 5.1 years for the observation and interferon groups, respectively (p = 0.09).  Grade 4 toxicities (neutropenia, myalgias, fatigue, depression, and neurologic untoward effects) occurred in 11.4% of interferon-treated patients.  These results show that adjuvant interferon alfa-NL treatment following nephrectomy did not provide clinical benefit for patients with locally advanced RCC. (Messing EM, et al. J Clin Oncol 2003;21:1214-1222)

 

MYELOFIBROSIS WITH MYELOID METAPLASIA (MMM)

Combination thalidomide plus prednisone therapy.  MMM is a clonal hematopoietic stem cell disorder characterized by progressive anemia, leukoerythroblastosis, splenomegaly, progression to acute leukemia, and premature death.  The lack of effective therapy for this disease has stimulated study of the pathogenetic mechanisms of MMM.  The demonstration of progressive neoangiogenesis in MMM led to investigation of the antiangiogenic agent thalidomide.  Single-agent thalidomide at doses >100 mg/day has been associated with an amelioration of the anemia of MMM in approximately 20% of cases, but is poorly tolerated.  Ruben Mesa and coworkers at the Mayo Clinic (Rochester, MN) utilized low-dose (50 mg/day) thalidomide in combination with a 3-month course of oral prednisone (0.5, 0.25, and 0.125 mg/kg/day for the first, second, and third months, respectively) to treat 21 patients with symptomatic MMM (blood hemoglobin concentration <10 g/dL or symptomatic splenomegaly).  Thalidomide plus prednisone was tolerated well and 20 (95%) patients completed 3 months of therapy.  Thirteen (62%) patients experienced improvements in anemia and 4 of 10 red blood cell transfusion-dependent patients became transfusion independent.  In addition, 6 of 8 patients had platelet responses and spleen size decreased by >50% in 4 (19%) patients.  Responses were reported to be durable following discontinuation of prednisone.  The clinical responses were not found to correlate with changes in intramedullary angiogenesis.  These results indicate that the combination of thalidomide plus prednisone is a potentially effective regimen for patients with MMM. (Mesa RA, et al. Blood 2003;101:2534-2541)

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